1
Augmenting immune surveillance and suppressing immune escape are crucial in tumor immunotherapy.
2
However, the function of JNK1 in tumor immune surveillance is unknown.
3
Effective immune surveillance is essential for maintaining protection and homeostasis of peripheral tissues.
4
Host immunity recognizes nascent malignant cells - a process referred to as immune surveillance.
5
These data suggest immune surveillance can be impaired by tumor-induced alterations in cDC development.
6
We conclude that NK cells can mediate immune surveillance of a low leukemia burden.
7
Tumor-induced immunosuppression is one of the crucial mechanisms of tumor evasion of immune surveillance.
8
Tumors are believed to emerge only when immune surveillance fails.
9
These insights provide new, visual information regarding immune surveillance and antigen presentation within the CNS.
10
However, how effector sequence polymorphisms enable pathogens to dodge host immune surveillance remains largely unknown.
11
Detection of microbial products by host inflammasomes is an important mechanism of innate immune surveillance.
12
Mast cells are central players in immune surveillance and activation, positioned at the host-environment interface.
13
Inflammation also affects immune surveillance and responses to therapy.
14
Leukocyte trafficking and recruitment is a critical process in host immune surveillance and in inflammatory diseases.
15
Natural killer T (iNKT) cells can help mediate immune surveillance against tumors in mice.
16
IFNs are cytokines with important antiproliferative activity and exhibit key roles in immune surveillance against malignancies.
