Immunoglobulin G specifically stained human Betz cells, whose axons are preferentially damaged.

Immunoglobulin G antibodies against H. pylori were measured by enzyme-linked immunosorbent assay.

Immunoglobulin G anti-HEV emerged on Day 67; subsequently, hepatitis was resolved.

Normal immunoglobulin G did not have such effects on retinal cells.

These nonspecific responses were transient and of the immunoglobulin G isotype.

Serum immunoglobulin G, A, and M were measured by end-point nephelometry.

Intrathecally produced immunoglobulin G was resolved into sharply focused, straight and easily identifiable fractions.

H. pylori infection was measured serologically by immunoglobulin G antibody titers against H. pylori.

Another patient had a below normal immunoglobulin G level.

BBB permeability was detected with sodium fluorescein extravasation and further confirmed by immunoglobulin G immunohistochemistry.

Serum samples were analyzed for immunoglobulin G antibodies against H. pylori by enzyme-linked immunosorbent assay.

One month after boosting at age 4 years, menC immunoglobulin G and SBA levels increased significantly.

The mechanisms involved in sequential immunoglobulin G (IgG) class switching are still largely unknown.

The pH gradients were stable and the inter-gel reproducibilities of individual immunoglobulin G patterns were good.

Immunoglobulin G Fc receptor IIb genetic variations were associated with early complete response in both groups.

Conjugate consisted of immunoglobulin G fraction of goat antiserum against chicken IgG bound to horseradish peroxidase.