1
Type 2 diabetes mellitus and impaired glucose tolerance are associated with antipsychotic treatment.
2
A presentation with cellulitis may be a clinical indicator of impaired glucose tolerance.
3
We conclude that IL-1β is a causal driver of impaired glucose tolerance in GDM.
4
A timely diagnosis of impaired glucose tolerance (IGT) is desirable in obesity.
5
Instead, long-term ozone exposure lowered circulating insulin and severely impaired glucose-stimulated beta-cell insulin secretion.
6
Results: Overabundance of TXNIP resulted in impaired glucose, insulin and pyruvate tolerance in normal mice.
7
However, impaired glucose and insulin homeostasis were still conserved in HFD-fed mice exposed to ES.
8
The prevalence of impaired glucose regulation increased with age.
9
Diabetes mellitus is a chronic metabolic disorder characterized by insulin deficiency and impaired glucose metabolism.
10
Conclusion: People with T1D have impaired glucose responses around a running competition compared to healthy individuals.
11
SAF has been suggested as a measure to screen for undiagnosed DM or impaired glucose tolerance.
12
Knockout of apoA-IV results in compromised insulin secretion and impaired glucose tolerance compared with WT mice.
13
Furthermore, elevated glucose concentrations in the sow during lactation may be suggestive of impaired glucose homoeostasis.
14
In obese rats the decrease of thyroid hormones level, impaired glucose toleranse, and dyslipidemia were detected.
15
Patients identified with impaired glucose regulation need specific treatment programs in order to prevent progression to diabetes.
16
Results: Pancreatic deletion of IL-1R leads to impaired glucose tolerance, a phenotype that is exacerbated by age.
