All but two ACA infarcts involved the internal capsule and basal ganglia.

Neither group showed cerebral cortex, corpus callosum, or internal capsule volume differences from control.

ADC values were obtained from white matter of the internal capsule and from the thalamus.

Right parahippocampus and left internal capsule showed greater mean diffusivity in early DAT compared with controls.

Magnetization transfer MRI revealed significant white matter abnormalities in the bilateral corpus callosum and internal capsule.

MR imaging showed a lesion of the left internal capsule with reduced ADC, which suggests an ischemic stroke.

Using an inversion recovery sequence, the myelination of the internal capsule was recorded as normal, abnormal or equivocal.

Girls with TS had lower FA in the inferior longitudinal fasciculus, right internal capsule and left cerebellar peduncle.

A reduction in volume and loss of phosphorylated neurofilaments in corpus callosum and internal capsule were observed on immunostaining.

Similarly, patients with catatonia showed also higher FA in the left internal capsule and left CST compared to healthy controls.

One of the affected areas, the left posterior limb of the internal capsule is linked to cognitive and executive functions.

Conclusions: Lesions of the insular cortex and the internal capsule are significantly associated with acute risk of aspiration after stroke.

Long-term symptom remissions were better following lesions of the anterior internal capsule and subcaudate white matter than of the cingulum.

The IBZ extended from the insular cortex to the internal capsule and the anterior part of the caudate nucleus head.

Results: All 4 cases with a normal internal capsule had a normal outcome in spite of the development of a PC.

The seizure group also had lower FA values in posterior limb of the internal capsule and the splenium of corpus callosum.