1
Cdx2 is an intestine-specific transcription factor required for normal intestinal epithelium development.
2
The intestinal epithelium undergoes a continual process of proliferation, differentiation and apoptosis.
3
However, the real meaning of Tβ4 reactivity in dysplastic intestinal epithelium remains unknown.
4
However, the role of STAT3 in the intestinal epithelium is not well understood.
5
The intestinal epithelium is a critical component of the gut barrier.
6
Central to EPEC-mediated disease is its colonization of the intestinal epithelium.
7
To test this, we study intestinal epithelium-specific IL-17RA-deficient mice in an immune-driven hepatitis model.
8
Then, we compared the mRNA levels between normal intestinal epithelium and neoplastic intestinal tissue.
9
CCL20 expression by the intestinal epithelium is also highly inducible in response to inflammatory stimuli.
10
The intestinal epithelium forms a barrier protecting the organism from microbes and other proinflammatory stimuli.
11
These findings indicate the importance of DGAT1 in fat metabolism and lipotoxicity in the intestinal epithelium.
12
Therefore, we investigated whether autophagy in the intestinal epithelium is involved in the pathogenesis of intestinal tumors.
13
Mechanisms that allow intestinal epithelium colonization, and host factors that favor AIEC persistence have been partly elucidated.
14
Lifelong self-renewal of the adult intestinal epithelium requires the activity of stem cells located in mucosal crypts.
15
Oral administration of nanoparticles is preferred but remains a challenge because transport across the intestinal epithelium is limited.
16
After 28 days of in vitro culture, hiPSC-derived progenitor cells differentiate into a monolayer of polarized intestinal epithelium.
