The patient presented with left hemiparesis due to lacunarinfarction.
2
A higher incidence of lacunarinfarction (LI) has been reported in nonwhite stroke populations.
3
Conclusions: Primary hematologic disorders and secondary hemostatic derangements may occur in patients with either cortical or lacunarinfarction.
4
We conducted a study on the added value of two D-dimer assays over clinical assessment for diagnosis of lacunarinfarction.
5
Conclusions: In patients with lacunarinfarction and diffuse white matter abnormalities, the presence of callosal atrophy may indicate cognitive impairment.
6
Patients with atherothrombotic infarction had higher total serum cholesterol and low density lipoprotein cholesterol concentrations than patients with lacunarinfarction.
7
Results: Five patients with lacunarinfarction and 7 patients with cortical infarction demonstrated raised fibrinopeptide A levels, indicating enhanced thrombin activity.
8
However, it is uncertain whether these abnormalities occur in both cortical and lacunarinfarction; this study aimed to further assess this issue.
9
Lupus anticoagulants were detected in 3 patients with lacunarinfarction and in 1 patient with cortical infarction.
10
Three patients in each group displayed decreased antithrombin III function, and 1 patient with a lacunarinfarction had a low protein C level.
11
Background and purpose: It is unclear why only some patients with lacunarinfarction and radiological evidence of diffuse white matter abnormalities have dementia.
12
Here we discuss the similarities between the genesis of pulmonary cavitation in various animal models and lacunarinfarction in the cerebral cortex of rats.
13
The presence of lacunarinfarctions was also associated with increased Abeta40 in both groups.