Meanings of late gadolinium in English

Infarct size and microvascular obstruction were assessed using late gadolinium enhancement imaging.

Infarct size was measured on day 1 by late gadolinium-enhanced CMR.

Eighteen subjects underwent pulmonary vein late gadolinium enhancement using the pencil-beam and projection navigator.

Nineteen percent had late gadolinium enhancement on cardiac magnetic resonance.

There was no evidence of late gadolinium enhancement (LGE) in the study population.

MIS was quantified on late gadolinium enhancement imaging as the proportion of left ventricular mass.

Abnormal late gadolinium enhancement (LGE) was detected in all 5 patients.

Cardiac MRI did not show any late gadolinium enhancement.

The significance of RV signal abnormalities during vasodilator stress perfusion and late gadolinium-enhanced CMR is yet uncertain.

A cardiac magnetic resonance imaging showed no significant late gadolinium enhancement to suggest infarct or myocardial scarring.

T2-weighted and postcontrast cardiovascular magnetic resonance scans were used to depict myocardial edema and late gadolinium enhancement, respectively.

No patient showed late gadolinium enhancement.

The severity index of late gadolinium enhancement was determined by scoring the extent of enhanced tissue in 30 myocardial segments.

The proposed method greatly reduces the inflow artifact in free-breathing pulmonary vein late gadolinium enhancement while allowing adequate respiratory motion compensation.

LA models with patient-specific geometry and fibrosis distribution were derived from late gadolinium enhanced magnetic resonance imaging of 6 AF patients.

Conclusion: The extent of myocardial fibrosis revealed by late gadolinium-enhanced MRI has a strong relationship to diastolic dysfunction in patients with HCM.