LV dysfunction is known to directly affect patient outcome in heart failure.

It appears that MVR could be considered in children before LV dysfunction develops.

Primary MR is a valvular disorder that induces LV dysfunction.

Two cases of LV dysfunction were simulated: Case A, systolic and Case B, diastolic dysfunction.

With further progression of LV dysfunction, the left atrium contribution to LV filling gradually decreased.

These results pave the way for approaches to prevent or treat LV dysfunction in obese patients.

Short-term follow-up demonstrated improvement in clinical parameters and longitudinal strain despite of residual regional LV dysfunction.

Further, the aged hearts had significant LV fibrosis and mild LV dysfunction compared with the young hearts.

Conclusions: Worse cognitive functioning can already be observed in early stages of LV dysfunction and heart failure.

Conclusion: Simple preoperative echocardiography measures allow the prediction of LV dysfunction after MVR in patients with leaflet prolapse.

Whether the moderately reduced myocardial Na,K-pump concentration is a pathogenetic factor in LV dysfunction remains to be determined.

Serial M-mode echocardiography revealed that cardiac amyloidosis was initially manifested as diastolic LV dysfunction rather than systolic dysfunction.

Conclusion: The mortality and morbidity of patients with LV dysfunction and FMR remain high despite current standard pharmacological therapy.

Conclusion: LV functional recovery was evident in majority of aortic stenotic patients with LV dysfunction after aortic valve replacement.

Measurement of Copeptin may provide prognostic information and the AVP system may be a therapeutic target in post-MI LV dysfunction.

Emergency cases and preoperative LV dysfunction cases were more frequently in Group C, but mortality did not differ among three groups.