Animal studies have shown that IGF-I is essential for mammary gland development.

The construction of the mammary gland is also peculiar in the Monotremes.

Thus, TIMP-3 is a critical epithelial survival factor during mammary gland involution.

Macrophages are required for proper mammary gland development and maintaining tissue homeostasis.

The mammary gland undergoes distinct periods of growth, development, and secretory activity.

Mastitis is a mammary gland inflammatory disease often due to bacterial infections.

Here, we reveal the dynamic nature of the nulliparous murine mammary gland.

We focus on three mammalian organs: hair follicle, tooth, and mammary gland.

Walker 256 mammary gland carcinoma cells were intratibially inoculated to induce BCP.

During mammary gland involution, total fibronectin and fibronectin fragment levels are increased.

We investigated whether Lgr4 functions in mammary gland development and mammary stem cells.

Our findings identify Igfbp7 as major regulator of involution in the mammary gland.

Adipose tissue in the mammary gland undergoes dramatic remodeling during reproduction.

We examined epithelial apoptosis in TIMP-3-deficient mice during mammary gland involution.

In contrast, ERRa overexpression increased breast cancer cell growth in the mammary gland.

Recent studies now suggest that a similar paradigm applies to the normal mammary gland.