However, the mechanisms controlling neuronal autophagy during starvation remain far from clear.

Infection of experimental animals with ZIKV causes significant neuronal damage and microgliosis.

However, the underlying mechanisms of how Aβ causes neuronal hyperexcitation remain unclear.

Learning causes a change in how information is processed by neuronal circuits.

Substance abuse in HIV-infected patients greatly influences the severity of neuronal damage.

The neuronal mechanisms underlying such maladaptive behavioral changes, however, are poorly understood.

Changes in cytosolic calcium are crucial for numerous processes including neuronal plasticity.

This strategy could improve neuronal survival through the provision of trophic support.

Technical challenges related to analyzing large-scale neuronal recording data are not trivial.

However, the mechanism of neuronal subtype specificity of disease phenotypes remains elusive.

Rho family GTPases have been implicated as important regulators of neuronal development.

However, no connexin has been documented at ultrastructurally defined neuronal gap junctions.

Cdk5 is a post-mitotic kinase with complex roles in maintaining neuronal health.

The ER stress response and neuronal apoptosis were attenuated by NGR1 treatment.

Thus, the physiological importance of orexin neuronal activity in nociception is unknown.

The molecular events surrounding injury-induced neuronal regeneration are beginning to be identified.