In addition, 24% of the sisters had hyperandrogenemia and regular menstrual cycles.
2
This familial aggregation of hyperandrogenemia in PCOS kindreds suggests that it is a genetic trait.
3
Its association with indices of insulin resistance and hyperandrogenemia is also seen in the same group.
4
We propose that hyperandrogenemia be used to assign affected status in linkage studies designed to identify PCOS genes.
5
Conclusions: Adult female acne may be triggered by diet, stress, and cosmetics and there is a distinct hormonal milieu that accounts for hyperandrogenemia.
1
No diagnoses of endometriosis or polycysticovaries, nothing like that?
2
Although 82% of women with recurrent early loss have polycysticovaries on ultrasound imaging, random serum LH concentrations are normal.
3
Twenty-one women with recurrent early pregnancy loss and 10 multiparous controls were investigated; 81% of them and one of ten control subjects had polycysticovaries.
1
Obesity in polycysticovarysyndrome aggravates the underlying predisposition towards insulin resistance.
2
Obesity increases the severity of the phenotype in women with polycysticovarysyndrome.
3
Only women with known primary infertility or severe polycysticovarysyndrome showed inadequate pregnancy rate.
4
Flutamide plus the oral contraceptive pill is beneficial for acne associated with polycysticovarysyndrome.
5
The majority of these women have polycysticovarysyndrome.
1
Obesity in polycysticovary syndrome aggravates the underlying predisposition towards insulin resistance.
2
Obesity increases the severity of the phenotype in women with polycysticovary syndrome.
3
Only women with known primary infertility or severe polycysticovary syndrome showed inadequate pregnancy rate.
4
Flutamide plus the oral contraceptive pill is beneficial for acne associated with polycysticovary syndrome.
5
The majority of these women have polycysticovary syndrome.
1
Conclusions: We identified metabolites that might have causal effects on PCOS development.
2
Environmental factors are thought to be involved in the development of PCOS.
3
Cardiovascular abnormalities represent important long-term sequelae of PCOS that need further investigations.
4
Based on the combination of PCOS symptoms, women formed seven outcome groups.
5
The present study aimed to explore the potential environmental risk factors of PCOS.
6
The number of GH pulses was not different between PCOS patients and controls.
7
However, the role of insulin secretory abnormalities in PCOS has received little attention.
8
Conclusion: PCOS is a complex disease that alone determines a deterioration of HRQoL.
9
However, premature coronary atherosclerosis has not been demonstrated in PCOS women.
10
PCOS has a strong heritable component based on familial clustering and twin studies.
11
The existence of an association between EDs and PCOS was proved.
12
One hundred sixty-nine PCOS patients and 338 matched controls were compared.
13
All women with PCOS in our study population were not obese.
14
Conclusion: Subclinical hypothyroidism does not influence the hormonal profile of women with PCOS.
15
No consistent association between PCOS risk and breast cancer was observed.
16
Conclusion: PCOS and related factors did not differ by sexual orientation.