1
Resting muscle concentrations of ATP and phosphocreatine did not change.
2
During cardioplegia, myocardial ATP and phosphocreatine contents increased and glycogen concentration tended to decrease.
3
Significant correlations were observed between endomyocardial and transmural adenine nucleotide and glycogen contents but not phosphocreatine content.
4
Creatine, a nitrogenous organic acid, replenishes cytoplasmic ATP at the expense of mitochondrial ATP via the phosphocreatine shuttle.
5
CKMT1 inhibition altered mitochondrial respiration and ATP production, an effect that was abrogated by phosphocreatine-mediated reactivation of the arginine-creatine pathway.
6
Transmural changes in left ventricular adenine nucleotide and glycogen levels were reflected in endomyocardial biopsies but those in phosphocreatine were not.
7
Low endomyocardial phosphocreatine levels are influenced by the biopsy technique, compromising the use of endomyocardial biopsies for establishing myocardial phosphocreatine content.
8
Dopamine also significantly attenuated the meningitis-induced reduction in both brain ATP and phosphocreatine levels and the increase in brain lactate level.
9
As the tumor volume increased from 60-1250 mm3, the ratio of phosphocreatine to inorganic phosphate systemically decreased.
10
We have also identified specific ways in which MuRF1 regulates metabolism at the level of the phosphocreatine shuttle and fatty-acid and glucose metabolism.
11
In conclusion, endomyocardial biopsies yield representative estimates of the average myocardial content of adenine nucleotides and glycogen but not of phosphocreatine in the normal heart.
12
However, hypoxia had no significant effects on regional brain levels of ATP and phosphocreatine or on intracellular pH, indicating successful adaptation to the hypoxic insult.
13
With extended delays between biopsy sampling and freezing, an increase in skeletal muscle phosphocreatine averaging 26% after 5 min was observed.
14
Phosphocreatine concentration was decreased and inorganic phosphate concentration was increased in soleus muscle frozen in situ.
15
Resting muscle concentrations of ATP and phosphocreatine did not change.
16
During cardioplegia, myocardial ATP and phosphocreatine contents increased and glycogen concentration tended to decrease.