We also identified several neurotransmitter receptors and postsynaptic density synaptosomal scaffold proteins.

Appropriate trafficking and targeting of glutamate receptors (GluRs) to the postsynaptic density is crucial for synaptic function.

We previously reported that NBCn1 interacts with the postsynaptic density protein PSD-95 in the brain.

This decrease is driven by an overall decrease in 2 postsynaptic density proteins, PSD-95 and SAP102.

For these spine profiles, the increased apposed Sv is due to the increased membrane area both at the postsynaptic density and beyond.

We show that peptide interference that prevents the localization of kalirin-7 to the postsynaptic density disrupts DOI-induced PAK phosphorylation and spine morphogenesis.

Asymmetric synapses were scored as a function of their position in the molecular layer, spine head size and shape, and postsynaptic density length.

In the electron microscope, labeled swellings form synapses that are symmetric (in that there is little postsynaptic density), a characteristic of inhibitory synapses.

The postsynaptic density (PSD) of central excitatory synapses is essential for postsynaptic signaling, and its components are heterogeneous among different neuronal subtypes and brain structures.

Therefore, NMDAR and nNOS are part of the intricate scenario of postsynaptic densities.

Spines with either perforated or nonperforated postsynaptic densities were contacted by EC axons.

The number of large, concave spine synapses with large postsynaptic densities significantly increases in the central zone of synaptic activation.