1
When primed mice were injected with lymphokine before secondary i.p.
2
Urinary excretion of (14)C-chloroform was higher in primed mice after administration of the lethal dose.
3
Neutrophil depletion in primed mice abrogated the protective effects of transferred macrophages and inhibited their in vitro binding to larvae.
4
These data suggest that higher detoxification may play a role in the lower initiation of kidney injury observed in primed mice.
5
In addition, the response is broad because the primed mice respond to an array of peptides in different major histocompatibility complex haplotypes.
6
Nonlethal infection of vaccine- primed mice generated secondary IgM and IgG antibody responses in serum and PLF and an IgA antibody response in PLF.
7
Vaccination of infection- primed mice boosted the IgM and IgG antibody concentrations in serum and PLF but had no effect on IgA antibody concentrations.
8
This temporal separation was much less apparent for the more rapid secondary response resulting from challenge of H3N2- primed mice with an H1N1 virus.
9
Exposure of primed mice to a lethal dose of chloroform led to 40% lower chloroform levels (AUC(15-360 min)) in the systemic circulation.