1
These findings indicate that the full proinflammatory response occurs in two stages.
2
Primary CMV infection induced a clear proinflammatory response that was maintained during latency.
3
Surgical necrosectomy induces a proinflammatory response and is associated with a high complication rate.
4
Background: Vitamin D is a potent inhibitor of the proinflammatory response and thereby diminishes turnover of leukocytes.
5
Variant TLR genotypes are associated with an increased circulating antigen burden and a decreased proinflammatory response in cirrhosis.
6
Our data suggest that the EAT environment is susceptible to microbial colonization that might stimulate a proinflammatory response.
7
These changes alter fluid and electrolyte homeostasis and result in an exaggerated proinflammatory response driven, in part, by infection.
8
Our results show that both extracts enhanced L. acidophilus-induced IFN-β production and slightly decreased the proinflammatory response to E. coli.
9
These findings support the hypothesis that COL17 plays an important regulatory role in the EK proinflammatory response, acting largely via NF-kappa B.
10
Implications: Chemotherapy treatment of pancreatic cancer-associated fibroblasts results in a proinflammatory response driven by stress-associated MAPK signaling that enhances tumor cell growth and invasiveness.
11
Conclusion: In patients with infected necrotizing pancreatitis, endoscopic necrosectomy reduced the proinflammatory response as well as the composite clinical end point compared with surgical necrosectomy.
12
Therefore, the combined activities of Cph1p and Efg1p are required for C. albicans to stimulate a proinflammatory response in endothelial cells.
13
These cytoplasmic nucleic acids activate a host defense program that elicits a coordinated proapoptotic and proinflammatory response involving caspase-3 and caspase-1 activation.
14
Coupled with the latter are promotion of lipid synthesis and proinflammatory responses.
15
Some but not all antimicrobial peptides can neutralize LPS-stimulated proinflammatory responses.
16
Neointima-forming mechanisms are controversial but possibly linked to excess proinflammatory responses and dysregulated Notch signaling.
