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1 Conclusion: This is the first report of a renin - producing ovarian serous cystadenocarcinoma.
2 Body weight and plasma concentrations of norepinephrine and renin did not change.
3 This article summarizes present knowledge about the cellular mechanisms of renin secretion.
4 It can be considered as a unique, purely renin - dependent form of hypertension.
5 The angiotensin converting enzyme is a key enzyme in the renin - angiotensin system.
6 Plasma renin activity increased significantly after both the single and repeated doses.
7 The vascular renin activity was also determined after treatments with ACE inhibitors.
8 Adult mutant kidneys showed the typical upstream renin expression after chronic stimulation.
9 Initially plasma inactive renin remained unchanged but after 105 min it decreased.
10 Chronic active immunization against renin decreases blood pressure markedly in normotensive marmosets.
11 A number of studies have reported contributions of the renin - angiotensin system to atherosclerosis.
12 These targets include the renin - angiotensin - aldosterone system as well as the sympathetic nervous system.
13 This effect may in part contribute to the hypotensive actions of renin inhibitors.
14 This woman produced one hundred times the normal amount of renin .
15 There was no increase in plasma renin activity with either compound.
16 Objective: To assess the usefulness of indexes derived from renal vein renin measurements.
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