No single allergen could be identified as responsible for the observed difference.

Selective inhibition of JNK reduced inflammatory cell egress into the airway lumen after single allergen exposure.

This was observed for each single allergen.

The role of JNK mitogen-activated protein kinase activation may be limited in the pathogenesis of bronchial hyperresponsiveness after single allergen exposure.

Analyzing patterns of allergic sensitization from multiple allergens reveals additional relevant associations with asthma than the study of a single allergen or total IgE.