1
Here, we used small interference RNA to decrease Ku86 protein levels in human cells.
2
A low CCT2 mMC model induced by treatment of small interference RNA was established.
3
Suppression of TMEM166 expression by small interference RNA inhibited starvation-induced autophagy in HeLa cells.
4
SREBP-1 overexpression and knockdown by small interference RNA influenced the abundance of endogenous FASN.
5
Furthermore, HDAC1 knockdown by small interference RNA stimulated uPA expression and cancer cell invasion.
6
Knockdown of TxnIP with small interference RNA suggested that TxnIP mediates the glucose-induced impairment of thioredoxin activity.
7
Pharmacological inhibition of enzyme activity, small interference RNA-mediated gene knockdown, and gene knock-out of MMP-3 all provide protection against ER stress.
8
Reduction of sphingosine 1-phosphate receptor 1 with small interference RNA significantly attenuates this transendothelial electrical resistance elevation.
9
Further specific SIRT1 inhibition with EX 527 or small interference RNA specific to SIRT1 reversed the effect of resveratrol on cisplatin-induced toxicity.
10
An approximately 50% reduction in the amount of Ku86 protein was achieved 72 hours after transfection with Ku86-specific small interference RNAs.
11
When expression of PIP5KIbeta was inhibited with small interference RNA in HeLa cells, expression of PIP5KIalpha was also reduced slightly, but PIP5KIgamma expression was increased.
12
Conclusions: Small interference RNA in silencing cox-2 gene expression can enhance significantly the radiosensitivity of esophageal cancer EC9706 cells.
13
Small interference RNA-mediated myosin VI knockdown in the LNCaP human prostate cancer cell line resulted in impaired in vitro migration and soft-agar colony formation.
14
Small interference RNA-mediated superoxide dismutase 2 or sprouty-2 reduction also increased reactive oxygen species formation and impaired APC migratory capacity.
