1
Fasting arterial glucose and splanchnic glucose output was similar in all groups.
2
In conclusion, ET-1 infusion causes reduced splanchnic glucose production due to reduced glycogen-derived glucose release.
3
The peak rise in blood glucose was directly proportional to the increase in splanchnic glucose output.
4
During low rate intravenous glucose infusion splanchnic glucose output decreased to a similar extent in all groups.
5
Total precursor uptake could account for 30% of the splanchnic glucose output in the diabetic group.
6
A secondary rise in splanchnic glucose output occurred at 150 minutes and coincided with a secondary increment in arterial glucose.
7
Splanchnic glucose output increased during exercise 3-4 fold above resting levels in both groups.