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T-lymphocytes play an important role in the pathogenesis of asthma.
2
Sunlight exposure has been shown to alter DNA methylation patterns across several human cell-types, including T-lymphocytes.
3
CD4+ T-lymphocytes are increased in number, express activation markers and cluster around RS cells.
4
There was no difference between the curves of CD4 T-lymphocytes raise of coinfected and monoinfected groups.
5
The influence of potentially suppressing monocytes and positive regulatory T-lymphocytes was eliminated using purified B-cell cultures.
6
T-lymphocytes, B-lymphocytes and macrophages were identified and counted.
7
Nevertheless, DR(+)IC had reduced capacity to capture antigens and elicited poor proliferation and interferon-gamma secretion by T-lymphocytes.
8
This results in a moderate developmental delay of both B- and T-lymphocytes at key V(D)J recombination dependent stages.
9
This type of infection, however, almost never causes illness, but it does cause the immune system to produce T-lymphocytes and antibodies.
10
To deplete the T-lymphocytes from bone marrow cells we use either monoclonal anti-T-cell antibodies and complement or T101 ricin A-chain immunotoxin.
11
HIV-specific cytotoxic T-lymphocytes (CTL) are believed to play a key part in the control of virus levels throughout HIV infection.
12
T-lymphocytes and EBV-transformed B-lymphocytes were obtained from peripheral blood of 24 FH patients and 24 control subjects.
13
Conclusion: This prospective study confirms that hepatitis C virus infection does not seem to be associated with impaired CD4 T-lymphocytes recovery after HAART.
14
GAS5 was identified by functional expression cloning and is necessary and sufficient for normal growth arrest in both leukaemic and untransformed human T-lymphocytes.