Peroxisome proliferators and thyroid hormones have a number of common metabolic effects.

This may provide an important regulation of IGF bioactivity by thyroid hormones.

PRL pattern was unchanged as well as serum levels of thyroid hormones.

Through the former, thyroid hormones stimulate mitochondriogenesis and thereby augment cellular oxidative capacity.

They also showed decreases in some inflammatory factors and thyroid hormones.

These medications can alter the synthesis, secretion, transport, or metabolism of thyroid hormones.

The slight mediating effects of maternal thyroid hormones in early gestation warrant further evaluation.

We review the effects of thyroid hormones on mitochondrial energetics and principally oxidative phosphorylation.

The thyroid hormones change the adrenergic reactivity of the myocardium.

The role of thyroid hormones on lipolysis in human subcutaneous adipose tissue was investigated.

It is also a precursor to adrenaline, thyroid hormones, and some types of estrogen.

To confirm hypothyroidism, plasma thyroid hormones and TSH were assayed.

Increased uncoupling appears to be responsible for some of the hypermetabolic effects of thyroid hormones.

There are, however, other avenues by which the body can be oversupplied with thyroid hormones.

No effect of thyroid hormones on cyclic AMP accumulation was seen in non-isoproterenol stimulated tissue.

Iodine is an essential component of the thyroid hormones.