Laboratory mice that have been produced from a genetically manipulated egg or embryo.
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Several lines of transgenic mice have been constructed to address this question.
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Similar injury in testosterone-treated transgenic mice resulted in amelioration of these effects.
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However, mild muscle atrophy in some of the transgenic mice was noted.
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These transgenic mice develop HCCs with different incidence, kinetics and histopathological features.
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On observation, LARGE transgenic mice were indistinguishable from the wild type littermates.
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To test this possibility, we constructed transgenic mice that contained mutant minigenes.
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Osteoclastic miR-34a-overexpressing transgenic mice exhibit lower bone resorption and higher bone mass.
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In Tax transgenic mice, PS-341 administered thrice weekly inhibited tumor-associated NF-kappaB activity.
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Furthermore, we show that the frog enhancer functions appropriately in transgenic mice.
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They might play some roles in tumors progression in the transgenic mice.
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These transgenic mice have delayed hair growth, underdeveloped ears, and shorter tails.
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Hence, these hemizygous transgenic mice were considered to be viable and healthy animals.
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Both transgenic mice showed partial protection toward Aldara-induced inflammation compared with control groups.
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Furthermore, gut dysbiosis was recently reported in Huntington's disease transgenic mice.
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However, MLDS-treated transgenic mice gradually normalized the metabolic parameters and survived.
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In another experiment, APPSw transgenic mice were used to model chronic Tat exposure.
