Eighteen patients with mid to severe aortic valve regurgitation were retrospectively evaluated.

Fifteen of the 30 patients with severe left-sided atrioventricular valve regurgitation underwent reoperation.

The most important reason for re-intervention in both groups was mitral valve regurgitation.

Aortic valve regurgitation is often present due to connective tissue disease of a bicuspid valve.

Preoperative transthoracic echocardiography showed diffuse hypokinesia of the left ventricle and mild mitral valve regurgitation.

Root technique, follow-up length, and preoperative aortic valve regurgitation were predictors of proximal aorta dilatation.

Aggressive application of truncal valvuloplasty methods should neutralize the traditional risk factor of truncal valve regurgitation.

Angiotensin-converting enzyme inhibitors may not alter left ventricular overload in pediatric patients with aortic valve regurgitation.

There was no difference in VPB between patients with mitral valve stenosis and mitral valve regurgitation.

An echocardiogram revealed mitral valve regurgitation with vegetation.

Objective: Congenital mitral valve regurgitation (MVR) is a rare disease occurring in infancy or childhood.

Fourteen patients exhibited left-sided atrioventricular valve regurgitation during follow-up; 8 of them remained stable with medication only.

The long-term benefit of angiotensin-converting enzyme inhibitors in pediatric patients with aortic valve regurgitation is under consideration.

The patient was a 37-year-old female with incomplete form of Behçet's disease combined with aortic valve regurgitation.

We review our experience addressing both the valve and the aortic root when correcting bicuspid valve regurgitation.

We sought to determine the outcome of severe residual left-sided atrioventricular valve regurgitation, either medically treated or reoperation.