In addition, β4 decreases the apparent number of charges per voltage sensor.

We demonstrated the β1-adrenoceptor involvement in our fatal case by immunohistochemical analysis.

However, ablation of TGF-β1 from T cells significantly inhibited mammary tumor growth.

No difference was found in TGF-β1 levels, comparing the three study groups.

Elsewhere, collectrin is involved in pancreatic β cell proliferation and insulin secretion.

We also tested the β-lactamase activities of each of the bacterial strains.

We found that exposure to βOHB had no effect on excitation-contraction coupling.

TGF-β1 is a critical immune regulator that is highly expressed by LCs.

Furthermore, IL-1β affects insulin sensitivity through tumor necrosis factor-independent and dependent pathways.

In addition, there was inactivation of GSK3β leading to decreased β-catenin phosphorylation.

A role of IncP-1β plasmids in Fe uptake processes has been revealed.

The AP2β intensity and localization were analyzed for correlation with patients' survival.

The binding of the AAb to β2ARs was verified by surface-plasmon-resonance analysis.

Ca v β 3 overexpression in human islets also impaired insulin secretion.

Plasma β-hydroxybutyrate concentration with the folic acid supplement followed the same tendency.

The crystal structures revealed that the lectins possess a seven-bladed β-propeller fold.