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The PDK said its coalition partner had broken the agreement.
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Here we address the molecular mechanisms underlying the regulation of protein kinase C betaII by PDK-1.
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Power from the engine and the rear motor hits the street through a seven-speed PDK gearbox.
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Our findings reveal new roles for cardiac metabolism and the PDK-PDC axis in cardiomyocyte behavior following cardiac injury.
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Throttle response is even more immediate because of the lightning fast shifts from the Porsche PDK automatic transmission.
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Furthermore, we find that PDK activity can modulate cell cycle progression and protrusive activity in mammalian cardiomyocytes in culture.
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Porsche has ditched the 7-speed PDK for an 8-speed gearbox.
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Coupled to an eight-speed PDK (dual-clutch) transmission, this new motor rewrites what was already a powerful yet usable drivetrain combination.
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One option left unchecked on this model is the much-vaunted Porsche Doppelkupplung (PDK) dual-clutch automatic transmission, and thank goodness for that.
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This review will focus on the regulation of PDK-1 and the various mechanisms which it uses to contribute to the activation of target kinases.
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Disruption of this interaction promotes the autophosphorylation of protein kinase C, suggesting that the binding of PDK-1 to the carboxyl terminus protects it from autophosphorylation.
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PDK-1-induced activation of a C-terminal truncation mutant of p70S6K was also enhanced by myr-PKCzeta.