Significados de alveolar epithelium en inglés

However, the effects of chronic alcohol abuse on the human alveolar epithelium are essentially unknown.

Conclusions: The pulmonary alveolar epithelium is the chief target of SARS-CoV, with macrophages infected subsequently.

What needs to be added to this picture is the glycocalyx of the alveolar epithelium.

Proliferation within alveolar epithelium increased as morphological grade increased.

Objectives: We aimed to determine the role of β-catenin in alveolar epithelium during bleomycin-induced lung fibrosis.

Among others, it proved the existence of a continuous alveolar epithelium and demonstrated the surfactant lining layer.

Stretch can modify cell viscoelastic properties, which may compromise the balance of forces in the alveolar epithelium.

This study investigated whether pretreatment with pentoxifylline would protect the alveolar epithelium or lung endothelium from injury.

In lungs from patients with cystic fibrosis, the alveolar epithelium is damaged with ulceration and subsequent regeneration.

The tissue elements that oxygen molecules have to cross are the alveolar epithelium, the interstitium and the capillary endothelium.

Bronchiolar epithelium from organizing pneumonia patients resulted only weakly positive for MIF while no evidence of MIF expression was reported for alveolar epithelium.

Its histological features, the benign proliferation of alveolar epithelium and septal mesenchyme, allow for its distinction from other benign lesions of the lung.

In the normal lung secretory component can be demonstrated in bronchial ciliated cells, bronchial gland serous cells, bronchiolar epithelium and hyperplastic alveolar epithelium.

Since no treatment is available to repair damaged epithelium and prevent lung fibrosis, novel approaches to induce regeneration of injured alveolar epithelium are desired.

These studies demonstrate new mechanisms by which the induction of the heat shock response protects the barrier function of the alveolar epithelium in ALI.

Perturbations in several molecular pathways mediate vulnerability of the alveolar epithelium to injurious agents, including the unfolded protein response, autophagy, mitophagy, and cellular senescence.