Significados de amyloid deposition en inglés

Diagnosis requires demonstration of amyloid deposition and confirmation of the fibril protein type.

Conclusions: The amount of amyloid deposition and white matter damage independently predicts cognitive impairment.

One of the three patients showed amyloid deposition at the intestine, kidney, and skin.

The effects of amyloid deposition and white matter damage were assessed using logistic regressions.

The organs with the highest severity of amyloid deposition were the stomach and intestine.

In the systemic forms, amyloid deposition is widespread and causes severe dysfunction of vital organs.

Although elderly patients often have amyloid deposition in the gastrointestinal tract, they are usually asymptomatic.

Conclusions: In this study, postoperative cognitive dysfunction was not associated with 6-week cortical amyloid deposition.

Electron microscopic examination showed "tubular" amyloid deposition intermingled with invasive carcinoma and granuloma.

Interpretation: Vascular health had quantifiably greater impact on neurodegeneration in AD regions than on amyloid deposition.

Results: Biopsy specimens demonstrated that the calcification in these tumors was contained in areas of amyloid deposition.

Background: Brain amyloid deposition is one of the main pathological characteristics of Alzheimer's disease (AD).

It is important that imaging studies be designed to reflect the slow nature of the process of amyloid deposition.

The relationship between cognitive dysfunction and regional amyloid burden and the rate of postoperative amyloid deposition merit further investigation.

APOE-ε4 carriers showed reduced covariance between two networks encompassing frontal and temporal regions, which constitute preferential target of amyloid deposition.

The importance of systemic workup and accurate tissue diagnosis in differentiating primary amyloidosis and secondary amyloid deposition will be highlighted.