Bone death caused when the bone no longer receives blood supply.
1
Our goal was to identify genetic and nongenetic risk factors for osteonecrosis.
2
In this small series, no patient has developed osteonecrosis of the jaw.
3
Three patients had osteonecrosis develop at the site of their radiation therapy.
4
In conclusion, osteonecrosis was associated with inherited variations near glutamate receptor genes.
5
The mechanism whereby lovastatin can counteract steroid-induced osteonecrosis and osteoporosis is poorly understood.
1
Conclusion: Clinically occult AVN of the hip is common in patients with SLE.
2
Medical records were reviewed for serologic and clinical variables that might predict AVN.
3
He developed AVN of bone after 2 years of treatment.
4
Decompression and grafting are safe and reasonably effective in retarding the progression of AVN.
5
These were compared to 55 hips with AVN treated nonoperatively.
1
One patient developed the extensive bone infarction of the left knee without the use of corticosteroids.
1
Purpose: To assess the signal changes of avascular bone necrosis after core decompression.
2
In addition to the infarction in the right tibia, severe bone necrosis was detected in components of the left hip.
1
Factors predisposing to avascular necrosis were delayed reduction and severity of trauma.
2
There was no evidence of avascular necrosis in any of the uninvolved hips.
3
One child had a stiff hip and a radiograph showed signs of avascular necrosis.
4
No patient developed avascular necrosis of femoral head after operation.
5
This could lead to avascular necrosis of the femoral head.
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