Significados de asthmatic mice en inglés

Petroleum ether, chloroform and n-butanol extracts lowered the Penh values of asthmatic mice.

All extracts decreased airway responsiveness in asthmatic mice compared with the untreated group.

Results: Foxp3 mRNA and protein were increased in the lung tissue of asthmatic mice.

ALB also alleviated AHR in asthmatic mice and improved pathological changes in the lungs.

Loss of function experiment was performed via transplantation of miRNA in OVA-induced asthmatic mice.

Eosinophilia cells in the serum of asthmatic mice were counted.

Objective: To observe the effects of artesunate on airway responsiveness and airway inflammation in asthmatic mice.

Conclusion: For asthmatic mice undergoing ozone exposures, AHR is not an accurate indicator of the severity of asthma.

Conclusion: Artesunate can improve airway hyperresponsiveness and airway inflammation in asthmatic mice and has a certain therapeutic effect on asthma.

Mice exposed to SiONPs showed markedly increased Penh values, inflammatory cell counts, and inflammatory cytokine levels compared to OVA-induced asthmatic mice.

Results: In this work, we have demonstrated for the first time the quantitative measurement of regional ventilation in normal and asthmatic mice.

The results showed that ozone could induce airway hyperresponsiveness (AHR) in controls and an additional enhancement of preexisting AHR in asthmatic mice.

The profile of CD9+ B cells was analyzed using blood from severe asthmatic patients and normal and asthmatic mice by flow cytometry.

We have found that lungs from asthmatic mice had sustained up-regulation of PD-1 and PD-L2, with PD-L2 primarily on dendritic cells.