Significados de axonal regeneration en inglés

Inhibitory molecules in CNS myelin affect axonal regeneration after injury.

Inhibitors in myelin play a major role in preventing spontaneous axonal regeneration after CNS injury.

This study developed a slice culture model system to study axonal regeneration after spinal cord injury.

Thus, NeuroHeal is a promising treatment for spinal nerve root injuries and axonal regeneration after trauma.

Spinal cord injury (SCI) is characterized by dramatic neurons loss and axonal regeneration suppression.

Neuronal damage and axonal regeneration inhibition are the main reasons to poor functional recovery after ischemia.

This is a prerequisite for successful axonal regeneration.

Several studies have suggested that growth factor-promoted axonal regeneration can occur for an extended period after injury.

Hematogenous macrophages remove myelin debris from injured peripheral nerves to provide a micro-environment conducive to axonal regeneration.

Ongoing efforts to promote axonal regeneration after SCI have involved multiple strategies that have been only partially successful.

Object: The glial scar composed of astrogliosis and extracellular matrix deposition represents a major impediment to axonal regeneration.

Of these cells, the olfactory ensheathing cells have become a more favorable candidate for extensive remyelination and axonal regeneration.

However, to be characterized definitively as inhibitory rather than nonpermissive, MAG must also inhibit axonal regeneration when presented in solution.

Various experimental strategies to improve neuronal survival, axonal regeneration and target reinnervation are described including pharmacological approaches and cell-based therapies.

Even when axonal regeneration is successful, the non-specific target reinnervation and the limited rebuilding of spinal circuits impair functional recovery.

Calcium is an important messenger in the neuronal system, but its specific role in axonal regeneration has not been fully investigated.