Significados de beta-oxidation en inglés

The first reaction of peroxisomal beta-oxidation is catalyzed by an acyl-CoA oxidase.

The new metabolites fall into two categories, glucuronides and phenylbutyrate beta-oxidation side products.

In wax ester fermentation, fatty acids are synthesized by reversing beta-oxidation in mitochondria.

In contrast, mitochondrial beta-oxidation rates of palmitate were doubled, despite the severely affected inner mitochondrial membrane.

Among these molecules, carnitine is an endogenous amine which is an essential cofactor for mitochondrial beta-oxidation.

L-Carnitine, a quaternary ammonium compound, plays an important role in beta-oxidation of fatty acids in mammals.

Under the same conditions, lignoceric acid beta-oxidation was impaired in the above three peroxisomal disease states.

Fatty acyl-CoA synthetase and fatty acid beta-oxidation activities were present in both the purified mitochondrial and peroxisomal enriched fractions.

Results: These mice showed fasting hepatic steatosis and diminished ketogenesis associated with decreased expression of genes involved in mitochondrial beta-oxidation.

Northern blot analysis revealed a decrease in the expression of peroxisome proliferator-activated receptor alpha-response genes involved in fatty acid beta-oxidation.

Previously, a beta-oxidation-blocked derivative of ATCC 20336 was constructed which showed a dramatic increase in the production of dicarboxylic acids.

Fatty acid beta-oxidation activities also separated as two distinct peaks, and the activities were highest in the peroxisomal enriched fractions.

Medium-chain acyl-CoA dehydrogenase (MCAD) deficiency is the most common inherited disorder of mitochondrial fatty acid beta-oxidation in humans.

Several enzymes of the beta-oxidation pathway have been shown to be induced after stimulation with peroxisomal proliferators, including several hypolipidemic drugs.

It was then hypothesized that decreases in peroxisome size could be paralleled by changes in peroxisomal beta-oxidation and estradiol catabolism actions.

Stearic acid beta-oxidation by normal fibroblast homogenates was several-fold greater compared to the oxidation of the two branched chain fatty acids.