Significados de bronchopulmonary dysplasia en inglés

Treatment with exogenous surfactant may decrease the incidence or severity of bronchopulmonary dysplasia.

Objective: Inconsistent effects of vitamin A supplementation on prevention of bronchopulmonary dysplasia have been reported.

Rate of bronchopulmonary dysplasia was similar in both groups.

Infants with bronchopulmonary dysplasia may develop pulmonary arterial hypertension.

One case of bronchopulmonary dysplasia was reported in each group in one study involving 29 infants.

AT2 cell injury has been implicated in several lung diseases, including idiopathic pulmonary fibrosis and bronchopulmonary dysplasia.

Objective: Oxidant injury and lung inflammation in extremely premature infants are associated with the development of bronchopulmonary dysplasia.

We conclude that treatment with BMDAC after neonatal hyperoxia restores lung structure in this model of bronchopulmonary dysplasia.

Recent technologic innovations and refinements have increased and enhanced the armamentarium that potentially can prevent or ameliorate bronchopulmonary dysplasia.

In infants with bronchopulmonary dysplasia, the influence of the severity of their pulmonary disease on neurodevelopmental outcome is unknown.

We report on severe bronchopulmonary dysplasia in a preterm infant who developed secondary, symptomatic and progressively severe pulmonary arterial hypertension.

Small for gestational age infants had increased risk of death or bronchopulmonary dysplasia regardless of their patent ductus arteriosus status.

By the time symptoms appear, it may already be too late to prevent a severe course, with bronchopulmonary dysplasia or mortality.

In patients with pulmonary hypertension and bronchopulmonary dysplasia, therapeutic strategies focus on the underlying chronic lung disease and use of vasodilators.

These data establish that an abnormal extracellular matrix without overt elastolysis is sufficient to confer susceptibility to postnatal normoxia, reminiscent of bronchopulmonary dysplasia.

Ad-infected neonates stayed longer in the neonatal intensive care unit, required more days of respiratory support and were more likely to develop bronchopulmonary dysplasia.