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C-kit positive cardiacprogenitor cells increased their numbers in exercise-induced physiological hypertrophy.
2
C-kit and Sca-1 positive cardiacprogenitor cells were activated by swimming training.
3
Conclusion: This study presents that swimming-induced physiological hypertrophy initiates activation of cardiacprogenitor cells.
4
In pregnancy, c-Kit positive cardiacprogenitor cells were activated.
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Immunofluorescent staining was used to compare the number of C-kit and Sca-1 positive cardiacprogenitor cells.
6
These proteins are indicated as potential targets to control cardiacprogenitor cell fate by materials design.
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Background: Endogenous cardiacprogenitor cells are a promising option for cell-therapy for myocardial infarction (MI).
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Collectively, these results permit enhancement of stepwise differentiation and facilitate isolation and expansion of cardiacprogenitor cells.
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Background: In recent years, resident cardiacprogenitor cells have been identified in, and isolated from the rodent heart.
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Our study provides evidence that receptor-kinase-dependent EphB4-forward signaling plays a crucial role in the development of cardiacprogenitor cells.
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Conclusion: This study presents that pregnancy-induced physiological hypertrophy activates cardiacprogenitor cells and thereafter protects against cardiac ischemia-reperfusion injury.
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Rationale: Myocardial function is enhanced by adoptive transfer of human cardiacprogenitor cells (hCPCs) into a pathologically challenged heart.
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Studies on cardiacprogenitor cells (CPCs) and their derived exosomes therapeutic potential have demonstrated only modest improvements in cardiac function.
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Sca-1 positive cardiacprogenitor cells were increased in LV and OFT in mice swimming for 3 weeks.
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It has also been known that cardiacprogenitor cells including islet1-positive cells do not contribute to the cardiac repair and regeneration in mammals.
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Here we demonstrate that the paralog proteins YAP and TAZ exert a crucial role in adult cardiacprogenitor cell mechano-sensing and fate decision.