Significados de chronic colitis en inglés

A few of these cases develop a chronic colitis.

These results provide genetic evidence demonstrating that epithelial cell death is crucial for the development of chronic colitis.

The rarity of small bowel lesions should be a warning to be cautious when classifying chronic colitis in small children.

Conclusions: GED ameliorates intestinal fibrosis in dextran sulfate sodium-induced chronic colitis in mice and regulates major profibrotic cellular and molecular mechanisms.

In the present report, we addressed whether and how epithelial cell death plays a role in the development of chronic colitis.

The aim of this study was to elucidate the therapeutic effect and mechanism of Nissle1917 using murine acute and chronic colitis models.

Thus, we presented the view that inflammation could induce the metabolic reprogramming and promote the progression from chronic colitis to colorectal cancer.

Conclusions: Germinated barley foodstuffs feeding promotes bacterial butyrate production and attenuated inflammation in both spontaneous and chronic colitis in HLA-B27 transgenic rats.

In this study, we have investigated the role of the chemokine receptor CXCR3 in the development of chronic colitis in a murine model.

In summary, our data suggest a critical regulatory role of TNF-R2 signaling for disease exacerbation in Th1-mediated chronic colitis.

Anti-TL1A antibody prevented chronic colitis and attenuated established colitis by down-regulation of both T(H)1 and T(H)17 activation.

Disorders in enteric bacteria recognition by intestinal macrophages (Mphi) are strongly correlated with the pathogenesis of chronic colitis; however the precise mechanisms remain unclear.

The galectin-4-mediated stimulation of CD4(+) T cells is shown to exacerbate chronic colitis and delay the recovery from acute intestinal injury.