Significados de cortical excitability en inglés

The level of cortical excitability has been shown to change during motor learning.

The significance of these subtle pHi-shifts for cortical excitability and plasticity is discussed.

Pathological alterations in visual cortical excitability may be one contributory mechanism.

The parameters aimed to reduce cortical excitability beyond the end of the TMS train.

Aims: To investigate whether there are any interhemispheric differences of motor cortical excitability in MDD.

One possibility is that homeostatic mechanisms that keep cortical excitability within a normal physiological range are impaired.

We uncovered no evidence suggestive of any subclinical abnormality of the motor cortical excitability in PD patients.

This shortening was associated with normalization of the voluntary triphasic EMG pattern and the pre-movement cortical excitability increase.

TFES training produced a significant increase in MEP magnitude throughout the stimulation range, suggesting an increase in cortical excitability.

In contrast, in patients with poor recovery, this increase in cortical excitability at higher CS intensities was not seen.

The purpose of this study was to compare the effects of three training paradigms on cortical excitability in healthy subjects.

The absence of changes in cortical excitability in patients with poor recovery supports the relevance of our findings for recovery.

In this study, we described TMS evoked potentials (TEP) input-output properties to provide new insights into regional cortical excitability.

To examine this hypothesis, we assessed the effects of language on cortical excitability of the hand muscle representation by transcranial magnetic stimulation.

While patients demonstrated no abnormalities in cortical excitability, motor facilitation during action observation, putatively reflecting mirror neuron activity, was reduced in schizophrenia.

Conclusions: In Parkinson disease, the ability of premotor-motor connections to increase motor cortical excitability is defective but restored to normal by dopaminergic medication.