1
The extracellular matrix glycoprotein tenascin is sparsely distributed in normal human dermis.
2
Oedema in the papillary dermis was evident occasionally in all clinical groups.
3
After 12 h, fluorescence was detectable in tumor cells in deep dermis.
4
In contrast, TGFβ expression in the upper dermis was significantly attenuated following treatment.
5
The cells made all the right proteins to attach to the underlying dermis.
6
Skin biopsy results showed large atypical lymphoid cells in the dermis.
7
Underneath, the cells in the dermis begin to lose their strength and flexibility.
8
Cutaneous mucinosis is an uncommon lesion characterized by mucin accumulation in the dermis.
9
In human skin the junction between the epidermis and dermis undulates.
10
A similar but weaker response was seen in the dermis.
11
Therefore, we propose that two somite-derived populations of dermis progenitor cells can be distinguished.
12
Skin biopsy revealed an invasion of eosinophils in the dermis.
13
Thus the antioxidant capacity of the human epidermis is far greater than that of dermis.
14
Six studies reporting both acellular dermis and submuscular outcomes were used to estimate relative risks.
15
Most places, we're right down to the original dermis.
16
Cell destruction did not involve other elements of the dermis and spared parasitized Schwann cells.
