Significados de experimental colitis en inglés

However, the pathogenic importance of specific VEGF-A isoforms during T-cell-mediated experimental colitis remains largely unknown.

The aim of this study was to determine the effect of EGF in experimental colitis.

This process is arrested during experimental colitis, resulting in the accumulation of TLR-responsive pro-inflammatory mφ.

The results support that CAI administration is effective in ameliorating experimental colitis and preventing colonic fibrosis.

In this article, we aim to examine the novel effects of β-sitosterol on murine experimental colitis.

Carbon monoxide (CO) has been reported to ameliorate colonic inflammation and improve experimental colitis.

We previously identified the transcription factor CUX1 as crucial for protecting against experimental colitis in mice.

However, the effect of local administration of LPS and OVA on experimental colitis in vivo remains unknown.

Conclusion: Zinc administration induces metallothionein synthesis but has little effect on the short-term course of experimental colitis.

Conclusions: These results show that IL-10 has a functional role in regulating colonic inflammation during experimental colitis.

The present results indicate that CINC may have a crucial role in initiating neutrophil infiltration in experimental colitis.

In the present study we investigated the antiinflammatory effects of DIM on experimental colitis and colitis-associated colorectal carcinogenesis.

We evaluated the effect of zinc supplementation at different doses on a model of experimental colitis in the rat.

This study analyzes the cellular sites of altered IGF-I and IGFBP-expression in large bowel of rats with experimental colitis.

Background: Increased production of proinflammatory cytokines is characteristic of both animal models of experimental colitis and human inflammatory bowel disease.

Taken together, our results indicate that Se deficiency worsens experimental colitis and promotes tumor development and progression in inflammatory carcinogenesis.