Significados de fat feeding en inglés

Moreover, TBHetλKO mice were protected from developing glucose intolerance during high- fat feeding.

Short-term high- fat feeding induces a proinflammatory state that may impact the blood vessel wall.

VWA8 protein and mRNA were elevated in mouse liver in response to high fat feeding.

Results indicate a reversible down-regulation of hypothalamic glutamatergic synaptic strength in response to short-term high- fat feeding.

Here, we present a comprehensive molecular systems biology study of hepatic responses to high- fat feeding in mice.

Despite the increased weight gain with high- fat feeding, glucose tolerance was significantly improved in the transgenic mice.

This study aimed to test the hypothesis that GPR40 plays a role in insulin secretion after high- fat feeding.

This study demonstrates an essential role for PKD1 in the β-cell adaptive secretory response to high- fat feeding in mice.

In juvenile mice, obesity increased adipogenesis, but in adults, this was only seen in vWAT after prolonged high- fat feeding.

Diminished whole-body glucose uptake resulting from high- fat feeding was reversed completely by exercise training, but only partially by food restriction.

Conclusions: GPR40 plays a role not only in fatty acid modulation of insulin secretion, but also in GSIS after high- fat feeding.

Also, kidneys play a role in regulating glucose production; denervated kidneys abrogate the effect of fat feeding to cause insulin resistance.

Genetic deletion studies revealed that adipocyte-derived IGF1 regulated perigonadal but not subcutaneous adipose tissue mass during high- fat feeding and the development of obesity.

After high- fat feeding for 2 wk, insulin-stimulated whole body glucose utilization was significantly decreased by 26%.

In this study, we used an animal model to determine the metabolic consequences of maternal prenatal stress and high- fat feeding on the developing offspring.

These findings identify an essential role for HSP72 in blocking inflammation and preventing insulin resistance in the context of genetic obesity or high- fat feeding.