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Examples for "hepatosteatosis"
Examples for "hepatosteatosis"
1
In our study, we detected mild hepatosteatosis in all hypothyroidism-induced rats.
2
However, the composition of liver LDPs and the role of their alterations in hepatosteatosis are not well-understood.
3
In conclusion, we found that hypothyroidism-induced rats had mild hepatosteatosis as opposed to the control group histopathologically.
4
Here, we summarize recent literature reporting dissociation of hepatosteatosis from insulin resistance in genetic mouse models and clinical studies.
5
A reduction in liver fibrosis and hepatosteatosis was evident in RB394-treated obese ZSF1 rats.
1
However, it may prevent worsening of hepatic steatosis associated with weight gain.
2
The fatty-acid saturation of the diet hardly affected hepatic steatosis or inflammation.
3
HCV-associated IR may also cause hepatic steatosis and resistance to antiviral treatment.
4
Moreover, exercise can effectively reduce hepatic steatosis independent of changes in body mass.
5
Studies are needed to determine whether treatment of hepatic steatosis reduces systemic inflammation.
1
Metabolic changes due to nonalcoholic fatty liver disease may explain this relationship.
2
An autopsy showed he suffered from a fatty liver and enlarged heart.
3
The mechanism determining the progression from fatty liver to steatohepatitis is unknown.
4
This observation suggests a novel mechanism for the pathogenesis of fatty liver.
5
The presence and severity of fatty liver was assessed by abdominal ultrasonogram.
6
Conclusions: We modeled the natural history of fatty liver for adult Japanese men.
7
Resolution of NAFLD was defined as absence of ultrasound criteria for fatty liver.
8
Background: Obesity-related fatty liver disease is linked to mitochondrial dysfunction and oxidative stress.
9
Methods: Mice were fed the Lieber-Decarli ethanol diet to induce alcoholic fatty liver.
10
Previously, we demonstrated that development of fatty liver depends on adipocyte GH signaling.
11
This model induced a fatty liver with focal necrosis and fibrosis.
12
Muc2 deficiency protected mice from HFD-induced fatty liver disease and obesity.
13
The pathophysiologic continuum of non-alcoholic fatty liver disease begins with steatosis.
14
The highest rate of fatty liver was seen in obese children (38%).
15
However, it is unclear whether leptin improves fatty liver independently of these metabolic improvements.
16
Abdominal obesity and dyslipidemia are the important risk factors for nonalcoholic fatty liver disease.