1
Latency between stimulus and onset of gamma oscillation was increased with cooling.
2
Cooling dramatically attenuated gamma oscillation and abolished epileptiform bursts in a reversible manner.
3
Cooling also suppresses transition from gamma oscillation to ictal bursting at higher stimulus intensities.
4
Tetanic stimulation at high intensity elicited not only gamma oscillation, but also epileptiform bursts.
5
We discuss the concept that gamma oscillation abnormalities in schizophrenia often occur in the background of oscillation abnormalities of lower frequencies.
6
Can we dissociate gamma oscillations related to unconscious learning and to conscious perception?
7
Explicit memory encoding, in particular, relies on enhanced gamma oscillations.
8
Induced gamma oscillations and evoked potentials were not systematically co-localized.
9
Stimulation of BF PV projection neurons preferentially generated time-locked gamma oscillations in frontal cortices.
10
However, circuit dysfunction persists, indicated by alterations in kainate-induced gamma oscillations and impaired nest building.
11
As a consequence, the firing of pyramidal neurons was desynchronized and gamma oscillations were impaired.
12
Subsequent gamma oscillations produced by attended stimuli were smaller than those produced by unattended, irrelevant stimuli.
13
In LO, attention increased the baseline level of gamma oscillations during the expectation period preceding the stimulus.
14
Our results thus suggest that the functional role of gamma oscillations depends on the area in which they occur.
15
The background of this study is a recent debate about the functional meaning of evoked and induced gamma oscillations.
16
Cooling from 34 to 21 degrees C reversibly abolished gamma oscillations in all slices tested.