Significados de global ischemia en inglés

However, the hippocampus is vulnerable to global ischemia, which may lead to atrophy.

Thus, caspase-2L is both translated and activated following transient global ischemia.

Context: Elevated temperature worsens injury in experimental focal and global ischemia and brain trauma.

Isolated sham control as well as hypertrophied guinea pig hearts were subjected to global ischemia and reperfusion.

During 150 seconds of global ischemia the dominant frequency of activation decreased, while after reperfusion it increased rapidly.

IPC consisted of 5 min global ischemia plus 10 min reperfusion.

The hearts were subjected to 30 min of global ischemia followed by 30 min of reperfusion.

Taken together, the data suggest that caspase-2 induction and activation are important mediators of delayed neuronal death following transient global ischemia.

The dentate neurogenesis and synaptogenesis that occur after ischemia may contribute to memory recovery after hippocampal injury caused by global ischemia.

All hearts were subjected to 20-min global ischemia (37 degrees C) and 30-min reperfusion.

IPC was introduced by three cycles of 5-min ischemia and 5-min reperfusion before the global ischemia.

The response of the beta-adrenoceptor transduction system to global ischemia for 40 min was investigated in isolated working heart of rat.

Hearts were subjected to 20 min global ischemia or hypoxia followed by 30 min reperfusion or reoxygenation.

Three days of continuous systemic administration of Slit following global ischemia significantly attenuated the delayed neuronal death of hippocampal CA1 pyramidal cells.

Relationships between contractile function and cytosolic metabolism were examined in mature and immature rabbit hearts during graded hypoperfusion, global ischemia, and reperfusion.

To begin to address this issue, we studied the expression of bcl-2, and bcl-x mRNA and protein after global ischemia in the rat.