Significados de human erythrocytes en inglés

Clinical malaria is associated with the proliferation of Plasmodium parasites in human erythrocytes.

In human erythrocytes, malaria parasites perforate their enclosing erythrocyte membrane shortly before egress.

Strains containing allele III preferentially agglutinated sheep erythrocytes, whereas strains containing allele II preferentially agglutinated human erythrocytes.

Shape changes and vesiculation were induced in intact human erythrocytes by gradually decreasing pH in the cell suspension.

Similar to a variety of nucleated cells, human erythrocytes activate a non-selective cation channel upon osmotic cell shrinkage.

Like aerolysin, the hybrid was far more active than alpha toxin against human erythrocytes and mouse T lymphocytes.

The metabolic behaviour of human erythrocytes has been investigated with particular regard to the effect of their oxygenation state.

Treatment of human erythrocytes with 1 U ml-1 haemolysin within minutes induces a non-selective cation permeability.

However, forskolin activation of sperm adenylate cyclase activity could be observed after reconstitution with the regulatory component-enriched membrane from human erythrocytes.

The authors give account of their study concerning the effect of paracetamol on the acid haemolysis, and glutathione content of human erythrocytes.

Heparin-like molecules occur naturally on the surface of human erythrocytes, where they may act as receptors for binding of merozoite surface proteins.

Potent inhibitory activities of the conjugates against S. suis adhesion to human erythrocytes were determined at low nanomolar concentration by HAI assay.

Furthermore, while human and great ape leukocytes both express alpha 2-6-linked Sias, only human erythrocytes have markedly up-regulated expression.

After invading human erythrocytes, the malarial parasite Plasmodium falciparum, initiates a remarkable process of secreting proteins into the surrounding erythrocyte cytoplasm and plasma membrane.

All of the strains examined caused type 3 fimbriae mediated agglutination of tannic acid treated human erythrocytes despite sequence variation in the mrkD-encoding adhesin gene.

Results showed that a specialized broth supplemented with trace minerals and lysed human erythrocytes and serum is required for the recovery of nonculturable H. pylori.