Significados de human myocardium en inglés

The EAT secretome enhances arrhythmia susceptibility and inotropy of human myocardium.

In end-stage failing human myocardium and during digitalis treatment this mechanism may be of major importance.

Herein, we report the identification of isl1+ cardiac progenitors in postnatal rat, mouse and human myocardium.

Data obtained using specific beta-adrenoceptor agonists showed good compatibility of hESC-cardiac myocytes with adult human myocardium in terms of beta-adrenoceptor response.

Whether this effect is specific to heart failure per se or is a generalized feature of normal human myocardium is unknown.

Background: Only limited data exist describing the histologic and noncardiomyocyte function of human myocardium in end-stage heart failure (HF).

We validated the technique on human cardiomyocytes either freshly isolated or selectively excised from fixed sections of human myocardium by Laser Capture Microdissection.

Our study demonstrates that the pharmacological profile of a beta(3)-AR agonist on the human myocardium cannot be extrapolated from usual animal models.

Therefore, we aimed to determine the effect of resistin on the function of human myocardium and how resistin contributes to the proarrhythmic effect of EAT.

This study investigated the role of KATP channels and PKC in preconditioning in human myocardium and whether KATP channels are activated via a PKC-dependent pathway.

Morphological changes in human myocardium associated with pressure overload-induced left ventricular hypertrophy were studied in 22 normal and 21 hypertrophic hearts obtained at autopsy.

Late Sodium Current is a Novel Target for Amiodarone: Studies in Failing Human Myocardium.