Significados de human platelets en inglés

The present study has examined the fine structure of freeze-fractured human platelets.

P-LPA added to human platelets caused aggregation without the need for ADP.

We recently demonstrated that smoking cessation causes temporary hyper-aggregability of human platelets.

The data provide evidence for the direct activation by thrombin of phospholipase D in human platelets.

We found that human platelets express 284 miRNAs.

The same agonist-specific reductions in platelet aggregation occur in human platelets treated with a LARG inhibitor.

Previous and recent studies from our group challenge expression and functionality of NOS in human platelets and erythrocytes.

We used a specific gas chromatography-mass spectrometry (GC-MS) method to detect NOS activity in human platelets.

Thus, we demonstrated that coronary risk factors are associated with an impairment of PDNO release by human platelets.

To obtain the miRNA expression profile, next-generation sequencing was performed on human platelets obtained from 10 healthy subjects.

Conclusion Cigarette smoking cessation temporarily enhances the collagen-stimulated release of phosphorylated-HSP27 from human platelets in the short term.

These results suggest that the intracellular CO2 hydration-dehydration reaction is involved in the activation of human platelets by thrombin.

The roles of fibrinogen and fibronectin were contasted in the responses of human platelets to Staphylococcus aureus and collagen.

We have investigated the release of protons from human platelets and platelet aggregation induced by the calcium ionophore, A 23187.

In the present study, we investigated the effect of DIDS on Na+ regulation of alpha 2-adrenoreceptors in human platelets.

Intact human platelets, terminally differentiated cells with no growth potential, were found to possess unusually high levels of tyrosine-specific protein phosphorylation.