Significados de ischemic myocardium en inglés

Stunning is an important mechanism in progressive dysfunction of the ischemic myocardium.

Monophosphoryl lipid A-induced delayed preconditioning preserves the ischemic myocardium and endothelial cells.

Alcohol administration demonstrated an increase in nuclear factor k-B in the ischemic myocardium.

Thus A1ARs in ischemic myocardium are presumably saturated by endogenous adenosine.

The enhancement of beta-adrenoceptors was not observed in the ischemic myocardium.

Conclusions: Intracoronary infusion of VEGF165 significantly improves blood flow to the ischemic myocardium.

Stem cell therapies are promising strategies to regenerate human injured tissues, including ischemic myocardium.

This study provides a mechanism by which alcohol may be cardioprotective in ischemic myocardium.

Protection of ischemic myocardium is an important unmet need in reperfusion therapy of acute myocardial infarction.

Myocyte apoptosis has been shown clinically to contribute to infarct size following reperfusion of ischemic myocardium.

G-CSF was markedly up-regulated in the ischemic myocardium.

Acute myocardial infarction should be diagnosed as early as possible for the appropriate management to salvage ischemic myocardium.

Conclusions: Ethanol supplementation increased arteriolar density and significantly improved myocardial perfusion and endothelium-dependent vasorelaxation in chronically ischemic myocardium.

Novel strategies by which to repair ischemic myocardium after myocardial infarction include the use of three-dimensional polymer scaffolds.

Northern blot revealed a twofold increase in ET-1 mRNA in the ischemic myocardium compared with the nonischemic myocardium.

We also observed that CSs from post-MI hearts engrafted in ischemic myocardium induced angiogenesis and restored cardiac function.