In PS cells, however, cell fusion occurred when VSV-infected Lcells were co-cultivated.
2
Addition of sSgIGSF-Fc inhibited the aggregation of Lcells overexpressing mSgIGSF in a dose-dependent manner.
3
A stably transformed mouse Lcell clone has been isolated which expresses and secretes hIGF-I.
4
Histamine decreased adhesion of Lcells expressing the human histamine receptor and E-cadherin to an E-cadherin-Fc fusion protein.
5
Both SKBR3- L and HCC1954- Lcells showed greater sensitivity to obatoclax-induced apoptosis than parental cells.
6
In contrast stimulation of CD40 had no effect on adhesion of L-selectin expressing NALM6- Lcells.
7
In mouse Lcells, steady-state levels of Ii mRNA are barely detectable in untreated cells but increase sharply upon IFN-gamma treatment.
8
Interestingly, we also found that the development of acquired resistance to lapatinib resulted in acquired sensitivity to TRAIL in SKBR3- Lcells.
9
In a second resistant model, HCC1954- Lcells, p-AKT levels were not decreased and these cells did not show enhanced sensitivity to TRAIL.
10
We next examined whether this interaction inhibited homophilic binding of mSgIGSF by aggregation assays using Lcells that did not express mSgIGSF.
11
The DR beta I and DR beta III genes were expressed, together with the DR alpha-chain, after transfection into mouse Lcells.
12
Histamine did not alter the adhesion to the E-cadherin fusion protein of Lcells expressing either the histamine receptor or E-cadherin alone.
13
A chimeric Pcdh2 in which the original cytoplasmic domain was replaced with the cytoplasmic domain of E-cadherin was expressed in mouse Lcells.
14
In cadherin-deficient Lcells, I-afadin was mainly localized at cell-cell contact sites, but ZO-1 was mainly localized at the tip area of cell processes.
15
Infection of mouse Lcells with VSV leads to the formation of polykaryocytes about 4 to 12 h p.i.
16
The chimeric alpha 4-EBNA1 genes resident in Lcells were induced by HSV-1 infection and were regulated as alpha genes.