Significados de melanocyte en inglés

Transient suppression of the proliferation of melanocyte stem cells prevents stress-induced hair greying.

These results indicate that several aspects of melanocyte function respond directly to estrogenic stimulation.

The melanocyte counts by HMB-45 and Melan A were significantly lower than in normal skin.

Subsequently, the retraction velocity of melanocyte dendrites was assessed.

The influence of endothelins on melanocyte function was assessed.

Northern and Western blot analyses confirmed that both genes encoded products that were melanocyte lineage proteins.

Instead, hair greying results from activation of the sympathetic nerves that innervate the melanocyte stem-cell niche.

The occurrence of oxidative stress has been proposed as a pathogenetic mechanism for melanocyte degeneration in vitiligo.

The melanocyte-associated antigen gp100 constitutes one of the most attractive targets for T-cell-based immunotherapy against malignant melanoma.

On split-dopa, melanocyte counts were 98 and 93 per field, respectively.

Analysis of genomic locations for transcripts revealed that the distribution of melanocyte genes is non-random throughout the genome.

Neonatal UVR alters melanocyte migration dynamics by inducing their movement upward out of hair follicles into the epidermis.

This led to regression of the patients' metastatic melanoma as well as to the onset of autoimmune melanocyte destruction.

Recent studies reveal that melanocyte stress generates damage-associated molecular patterns that activate innate immunity, thus connecting stress to organ-specific inflammation.

Here we report that, in mice, acute stress leads to hair greying through the fast depletion of melanocyte stem cells.

In this study, we investigated the effects of TGF-beta1 on melanogenesis using a spontaneously immortalized mouse melanocyte cell line, Mel-Ab.