The 24-OHC levels were similar between the atorvastatin-treated subjects and matched controls.
2
Particularly, OHC and THC exerted superior hepato-protective and antioxidant activities to CUR.
3
OHCs have an amplification effect on the coding of low-frequency ultrasonic intensities.
4
Regulation of Aβ metabolism was explored as the pathogenic mechanism of 27-OHC.
5
Results: Treatment with 27-OHC resulted in severe pathologies in the ileum and colon.
6
It is still a matter of debate whether OHCs perform exocytosis.
7
These results provide baseline information on prenatal OHC background levels in the Mediterranean area.
8
In addition, increased amyloid plaques were also found in the hippocampus of 27-OHC-treated mice.
9
Gelatinolytic activity in conditioned cultured medium of OHC was accessed by a fluorescent substrate assay.
10
Cochlear outer hair cells (OHCs) terminally differentiate prior to the onset of hearing.
11
These results suggest that prestin may interact physically with CFTR in the lateral membrane of OHCs.
12
Postnatal expression of prestin and activity of OHCs may contribute to the maturation of hearing in rodents.
13
Outer hair cells (OHCs) are innervated by type II afferent fibers of as yet unknown function.
14
Meanwhile, co-treatment with ANS reduced intestinal inflammation and partially preserved intestinal barrier integrity in the presence of 27-OHC.
15
Prestin is the motor protein expressed in the cochlear outer hair cells (OHCs) of mammalian inner ear.
16
These results suggest that 27-OHC is deleterious for synaptic structural and functional plasticity, which may partially account for its neurotoxic effects.