Both polymorphonuclearcell infiltration and increased epithelial apoptosis are seen in gastric mucosa in the presence of Helicobacter pylori infection.
2
However, the chemotactic response of both polymorphonuclearcells and T lymphocytes in response to CXCL12 is increased.
3
H. pylori colonization is followed by infiltration of the gastric mucosa by polymorphonuclearcells, macrophages and lymphocytes.
4
The outcome of CDAD was not influenced by the number of circulating polymorphonuclearcells and CD4+ cells.
5
Polymorphonuclearcells (neutrophils) play an important role in the systemic inflammatory response syndrome and the development of sepsis.
6
Marked infiltration of the cornea by macrophages was observed, as was infiltration by polymorphonuclearcells, although to a lesser extent.
7
Polymorphonuclearcells predominated in 73 of 114 cases.
8
The combined administration of ST 789 with human peripheral blood polymorphonuclearcells completely inhibited the in vivo tumorigenicity of the T lymphoma.
9
EBC-46 induced oxidative burst from purified human polymorphonuclearcells, which was prevented by the Protein Kinase C inhibitor bisindolylmaleimide-1.
10
Large numbers of polymorphonuclearcells and monocytes invade the cornea by 24hr after surgery and persist for over 1 week.
11
Specimens examined included peripheral blood polymorphonuclearcells for CMV viremia and antigenemia determination, together with the most appropriate clinical samples when organ involvement was suspected.
12
Histopathological examination of tumor tissue from CPV2.NS1 treated group revealed infiltration of mononuclear and polymorphonuclearcells with increased extra cellular matrix, indicating signs of regression.