Affected animals show tremor, seizures, and die at about 24 postnatal days.

Marked QT segment anomalies on the electrocardiogram during the first postnatal days resolved completely within 3 weeks.

AC1 protein increased in abundance in the neonatal hippocampus from postnatal days 7-14.

At early postnatal days, brains displayed abnormal ventricular size and hemispheric asymmetry due to altered brain water homeostasis.

The anxiety-like behavior was evaluated by elevated plus-maze test on postnatal days 21 and 28.

Echocardiograms were performed on postnatal days 6 or 7.

Treatment lasted from postnatal days 0 to 11.

In this study, we evaluate oxidative stress in the hippocampus on the 1st and 30th postnatal days.

Phenotypic characterizations of the WT and KO mice were performed at postnatal days 1, 2, 4, and 6.

In rat ventricle, It is very small at birth and then increases markedly between postnatal days 8 and 20.

Methods: We recorded ultrasonic vocalizations from NS-Pten wild-type and knockout male and female mice on postnatal days 8 and 11.

Analysis performed at postnatal days 30, 90, 150, and 365 encompassed body mass and food intake monitoring and serum biochemistry determination.

From May 2011 through December 2015, no infant was treated with indomethacin until at least 8 postnatal days (conservative epoch).

Here we identified electrically coupled pairs of neurons between postnatal days 12 and 18 in rat visual cortex, somatosensory cortex, and hippocampus.

Heart UCP2 mRNA expression was low during the early postnatal days followed by a rapid and significant increase in the 2nd postnatal week.

The cumulative morphine dose-response was assessed on postnatal days 20 and 49, and stress-induced analgesia was assessed on postnatal days 29 and 49.